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Printable Handouts
Navigable Slide Index
- Introduction
- Acute pancreatitis (AP)
- Incidence of acute pancreatitis (1)
- Incidence of acute pancreatitis (2)
- Etiology of acute pancreatitis
- Outcomes of acute pancreatitis
- Etiology effect on severity of acute pancreatitis
- Pathophysiology of acute pancreatitis
- Diagnosis of acute pancreatitis
- Laboratory analysis
- Differential diagnoses of AP
- Chest X-ray
- Abdominal ultrasound
- Abdominal X-ray
- CT Severity Index (CTSI)
- EUS – Lithiasis and microlithiasis
- Magnetic Resonance Imaging (MRI)
- MRCP
- Criteria for severity prediction
- Ranson criteria
- APACHE II score
- Bedside Index of Severity in Acute Pancreatitis (BISAP)
- Grades of severity
- 2012: Revision of the Atlanta classification (1)
- 2012: Revision of the Atlanta classification (2)
- Local complications
- Grades of severity
- The determinant – based classification
- Comparing classifications (1)
- Comparing classifications (2)
- Classification of acute pancreatitis
- Outcomes of acute pancreatitis
- Two peaks of mortality
- Recognition & treatment of etiology
- Management of acute pancreatitis
- Pharmacological treatment
- Treatment of organ failure
- Analgetic treatment (1)
- Analgetic treatment (2)
- NSAID, opioids, epidural analgesia vs. control (1)
- NSAID, opioids, epidural analgesia vs. control (2)
- Epidural analgesia
- Parenteral hydration (1)
- Parenteral hydration (2)
- Ringer lactate vs. normal saline
- Is nutrition so important?
- ESPEN guidelines on clinical nutrition in acute pancreatitis
- Enteral nutrition vs. no nutritional support
- Immunonutrition
- Immunonutrition vs. other EN, TPN/no nutrition –mortality (1)
- Immunonutrition vs. other EN, TPN/no nutrition –mortality (2)
- Immunonutrition vs. other EN, TPN/no nutrition – infectious complication
- Immunonutrition vs. other EN, TPN/no nutrition – organ failure
- Effect of probiotics in acute pancreatitis
- Effect in severe acute pancreatitis
- Role of ERCP in acute pancreatitis management
- ERCP – therapeutic indications
- Role of ERCP in management of acute pancreatitis
- Antibiotic prophylaxis in AP
- Antibiotic prophylaxis in acute pancreatitis
- Is prophylactic use of antibiotics protective in severe acute pancreatitis? (Mortality)
- Is prophylactic use of antibiotics protective in severe acute pancreatitis? (Infected pancreatic necrosis)
- Is prophylactic use of antibiotics protective in severe acute pancreatitis? (Infected pancreatic necrosis – imipenem)
- Antibiotic prophylaxis or not?
- Antibiotic prophylaxis not recommended
- Local complications – Tx
- Pancreatic collections
- Walled-off necrosis (WON)
- Recommendations
- Infected pancreatic necrosis
- SEMS vs. plastic stents (1)
- Plastic vs. metal stents
- SEMS vs. plastic stents (2)
- Invasive interventions in AP (1)
- Invasive interventions in AP (2)
- Prevention of IAH in patients with SAP
- Nonsurgical treatment of IAH in SAP
- Conclusions
Topics Covered
- Acute pancreatitis
- Incidence and etiology
- Differential diagnoses
- Criteria for severity prediction
- Revision of the Atlanta classification
- Determinant-based classification
- Outcomes of acute pancreatitis
- Pharmacological treatment
- Immunonutrition
- Antibiotic prophylaxis
- Local complications
- Invasive interventions
Links
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Therapeutic Areas:
Talk Citation
Štimac, D. (2024, May 30). Acute pancreatitis [Video file]. In The Biomedical & Life Sciences Collection, Henry Stewart Talks. Retrieved December 5, 2024, from https://doi.org/10.69645/JLXB2114.Export Citation (RIS)
Publication History
Financial Disclosures
- Davor Štimac, MD PhD, graduated from the Faculty of Medicine, University of Rijeka, Croatia, in 1987, followed by a Master's degree in 1991, and PhD in 1997. He specialised in internal medicine with a sub-speciality in gastroenterology at the Rijeka Clinical Hospital Center and obtained the title of full professor in 2006. Currently he is working as a gastroenterologist and CEO in the private Special Hospital “Medico” and as full professor at the University of Rijeka. He is the former president of the Croatian Gastroenterology Society, the current first vice-president of the Croatian Academy of Medical Sciences, president of the Croatian Society for obesity and the vice-president of the Croatian Society for Quality in Health Care, as well as the national coordinator of the scientific-professional Committee for career development of young researchers. He has also received professional training at the University Hospital Udine (1995), Liver Research Institute, King's College Hospital, London (1997), University Hospital Padova, Italy (2000), University Hospital Copenhagen, (2003), San Raffaele Hospital, Milan (2004) and the Cleveland Clinic USA (2010). He is a member of the Board and past president of the European Society for Gastroenterology, Endoscopy and Nutrition (EAGEN), member of the United European Gastroenterology (UEG) Education Committee, President of the Manpower Committee of the European Board for Gastroenterology and Hepatology (EBGH) and the Editor of the Cochrane Hepato-Biliary Group. He has published more than 150 full papers in CC/SCI, many chapters and medical books and has been an invited lecturer at more than 150 gastroenterological symposium and congresses.
A selection of talks on Clinical Practice
Transcript
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0:00
I am Davor Stimac, Professor
of Gastroenterology.
I come from Rijeka, Croatia.
Today I will talk about
acute pancreatitis.
0:17
Acute pancreatitis by definition
is an inflammatory process of
the pancreas with
variable involvement
of other regional tissues
or remote organ systems.
0:32
According to the incidence
of acute pancreatitis,
today global incidence
with the data taken from
204 countries is
33.7 per 100000.
On this world map, you can see
that it differs from
country to country
from some countries
where it is eight per
100000 up to those where it
is nearly 200 per 100000.
1:10
According to meta-analysis
over the last 56 years
published in 2022 by
Iannuzzi and co-authors,
we can see that the
global incidence of
acute pancreatitis is
increasing at 3% per year.
1:31
If we're talking
about the etiology of
acute pancreatitis
at the first place,
the most common
etiology is biliary.
More than 50% of patients have
biliary cause of
acute pancreatitis.
Roughly 30% have alcohol as
an etiological cause
of acute pancreatitis
and 20% have some
other etiologies
like hypertriglyceridemia
and some others.