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Printable Handouts
Navigable Slide Index
- Introduction
- Incidence of acute pancreatitis (1)
- Incidence of acute pancreatitis (2)
- Etiology
- Severity of acute pancreatitis
- Pathophysiology of acute pancreatitis (1)
- Pathophysiology of acute pancreatitis (2)
- Pathophysiology of acute pancreatitis (3)
- Pathophysiology of acute pancreatitis (4)
- Diagnosis
- Laboratory analysis
- Radiographs of the abdomen
- Chest X-ray
- Transcutaneous ultrasonography
- Computed Tomography (CT)
- CT Severity Index (CTSI)
- Magnetic Resonance Imaging (MRI)
- MRI (examples)
- EUS of stones & microstones
- Magnetic Retrograde Cholangiopancreatography
- Differential diagnoses of AP
- Criteria for severity prediction
- Ranson criteria
- APACHE II score
- Diagnosis of acute pancreatitis
- 2012: Revision of the Atlanta classification (1)
- 2012: Revision of the Atlanta classification (2)
- 2012: Revision of the Atlanta classification (3)
- 2012: Revision of the Atlanta classification (4)
- 2012: Revision of the Atlanta classification (5)
- The determinant-based classification
- Comparison of classifications (1)
- Comparison of classifications (2)
- Classification of acute pancreatitis
- Outcomes of acute pancreatitis
- Pancreatic exocrine function
- Mortality of acute pancreatitis
- Two peaks of mortality
- World mortality
- Management of acute pancreatitis
- Pharmacological treatment in acute pancreatitis
- Role of ERCP in the management of AP
- ERCP: therapeutic indications
- Early ERCP vs. conservative therapy
- Enteral nutrition in AP
- Is nutrition so important?
- Nutrition in acute pancreatitis (1)
- Nutrition in acute pancreatitis (2)
- Frequency of EN administration
- EN vs. TPN in severe acute pancreatitis (1)
- EN vs. TPN in severe acute pancreatitis (2)
- EN vs. TPN in severe acute pancreatitis (3)
- EN vs. TPN in severe acute pancreatitis (4)
- Comparison of different EN formulations
- Immunonutrition vs. other EN, TPN, or no nutrition
- Probiotic prophylaxis in predicted severe AP
- Probiotics vs. other EN, TPN, or no nutrition
- Nutrition in acute pancreatitis
- Enteral nutrition vs. no nutritional support
- Early vs. on-demand EN administration (1)
- Early vs. on-demand EN administration (2)
- Early vs. on-demand EN administration (3)
- Enteral nutrition in AP
- Antibiotic prophylaxis
- Correlation of pancreatic necrosis and infection
- Prophylactic antibiotics in AP
- Is prophylactic use of antibiotics protective?
- Do we need more meta-analytic studies?
- Infected necrosis in AP
- Infected pancreatic necrosis: early diagnosis
- Minimally invasive techniques
- Percutaneous drainage for pancreatic necrosis
- Endoscopic therapy for pancreatic necrosis
- Laparoscopic technique for pancreatic necrosis
- Retroperitoneal approach for pancreatic necrosis
- Surgical necrosectomy
- Surgery in acute pancreatitis
- The role of early surgery in FAP
- Epidemiology of IAH and ACS in SAP
- Prevention of IAH in patients with SAP
- Nonsurgical treatment of IAH in SAP
- Surgical decompression of IAH in SAP (1)
- Surgical decompression of IAH in SAP (2)
- Pharmacological prevention still impossible
Topics Covered
- Epidemiology and etiology
- Pathophysiology of acute pancreatitis
- Diagnostic methods
- Revision of the Atlanta classification
- Severity prediction
- Outcomes of acute pancreatitis
- Therapeutic options
Links
Series:
Categories:
Therapeutic Areas:
Talk Citation
Štimac, D. (2017, August 31). Acute pancreatitis [Video file]. In The Biomedical & Life Sciences Collection, Henry Stewart Talks. Retrieved December 21, 2024, from https://doi.org/10.69645/YOWE7898.Export Citation (RIS)
Publication History
Financial Disclosures
- Davor Štimac, MD PhD, graduated from the Faculty of Medicine, University of Rijeka, Croatia, in 1987, followed by a Master's degree in 1991, and PhD in 1997. He specialised in internal medicine with a sub-speciality in gastroenterology at the Rijeka Clinical Hospital Center and obtained the title of full professor in 2006. Currently he is working as a gastroenterologist and CEO in the private Special Hospital “Medico” and as full professor at the University of Rijeka. He is the former president of the Croatian Gastroenterology Society, the current first vice-president of the Croatian Academy of Medical Sciences, president of the Croatian Society for obesity and the vice-president of the Croatian Society for Quality in Health Care, as well as the national coordinator of the scientific-professional Committee for career development of young researchers. He has also received professional training at the University Hospital Udine (1995), Liver Research Institute, King's College Hospital, London (1997), University Hospital Padova, Italy (2000), University Hospital Copenhagen, (2003), San Raffaele Hospital, Milan (2004) and the Cleveland Clinic USA (2010). He is a member of the Board and past president of the European Society for Gastroenterology, Endoscopy and Nutrition (EAGEN), member of the United European Gastroenterology (UEG) Education Committee, President of the Manpower Committee of the European Board for Gastroenterology and Hepatology (EBGH) and the Editor of the Cochrane Hepato-Biliary Group. He has published more than 150 full papers in CC/SCI, many chapters and medical books and has been an invited lecturer at more than 150 gastroenterological symposium and congresses.
A selection of talks on Clinical Practice
Transcript
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0:00
The following presentation is a short survey of the basic facts of
Acute Pancreatitis as well as to be introduced to curing Acute Pancreatitis.
0:13
Acute Pancreatitis is a rather common gastroenterological disease with incidence rating
from 5 to 80 cases per 100,000 inhabitants per year.
0:27
As shown in this slide and proven by many national studies,
the incidence of Acute Pancreatitis differs across Europe,
being the highest in the Scandinavian countries namely Finland.
The exact cause is not known but it's probably
related to the differences in the patients genetic traits,
social backgrounds and psychological behavior.
0:55
The etiology of acute pancreatitis is well defined.
Still in around 10 percent of patients,
the cause cannot be determined and these cases are categorized as idiopathic.
In more than 70 percent of the patients,
Acute Pancreatitis is caused either by biliary stones or alcohol consumption.
1:20
In most patients, Acute Pancreatitis is self-limited,
mild to moderate disease.
Complications are rare and death occurs in less than 1% of patients.
Unfortunately, in around 20% of patients,
Acute Pancreatitis follows a more severe course
with the development of Pancreatic Necrosis.
In these patients, infected Necrosis occurs in
30 to 40 percent of cases thus rising the mortality rate up to 30 percent.