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- Epidemiology
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2. Epidemiology of obesity
- Prof. Jacob Seidell
- Etiology and Causes
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3. Metabolic, adipose tissue and muscle predictors of obesity
- Prof. Ian Macdonald
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4. Neural control of food intake and energy balance
- Prof. Hans-Rudolf Berthoud
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6. Dietary determinants of obesity
- Prof. Arne Astrup
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7. Physical inactivity as a determinant of obesity
- Prof. Wim Saris
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8. Environmental causes of obesity: sociocultural, built environment and economic factors
- Prof. Shiriki Kumanyika
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9. The developmental origins of obesity
- Prof. David Barker
- Consequences of Obesity
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10. Obesity, dyslipoproteinemia and inflammation
- Prof. Robert Eckel
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11. Pathophysiology and management of obesity related hypertension
- Prof. Arya Sharma
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12. Disorders of overeating
- Prof. Albert Stunkard
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13. Obesity, weight loss and health-related quality of life
- Prof. Aila Rissanen
- Dr. Jarmo Kaukua
- Treatment Options
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14. Lifestyle modification for weight control
- Prof. Thomas Wadden
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15. Bariatric / obesity surgery
- Prof. Henry Buchwald
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16. Can obesity be prevented in the current obesogenic environment?
- Prof. Boyd Swinburn
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17. Pharmacotherapy for obesity: why it is needed
- Prof. Joe Proietto
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18. Pharmacotherapy for obesity: hunger suppressors
- Prof. Joe Proietto
- Latest Developments in the Field
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19. Body composition
- Dr. Steven Heymsfield
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20. Adipose tissue metabolism and obesity
- Dr. Max Lafontan
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21. Obesity, present and future therapies
- Prof. Sir Stephen Bloom
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22. Energy expenditure in the lean and obese
- Prof. Dale Schoeller
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23. Obesity and adiponectin
- Prof. Philipp Scherer
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24. Ectopic fat: causes, consequences and treatment
- Prof. Steven Smith
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25. Adipose-immune interactions in obesity
- Dr. Vishwa Dixit
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26. Health benefits of intentional weight loss
- Prof. Xavier Pi-Sunyer
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27. Childhood obesity: implications for current and future health
- Prof. Peter T. Katzmarzyk
- Archived Lectures - These lectures may not cover the latest advances in the field
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29. Gastrointestinal peptides and food intake regulation
- Prof. Sir Stephen Bloom
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30. Obesity and mortality: questions and controversy
- Prof. David Allison
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31. Obesity, diabetes and the cluster of the metabolic syndrome
- Prof. Peter Wilson
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33. Health economics of obesity: new insights
- Dr. Anne Wolf
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34. Bariatric surgery: techniques and mechanisms of action
- Prof. Walter Pories
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35. Childhood obesity: implications for future health
- Prof. Peter T. Katzmarzyk
Printable Handouts
Navigable Slide Index
- Introduction
- Lipid and lipoprotein metabolism
- HDL metabolism
- Obesity,dyslipidemia,inflammation and ASCVD
- Obesity lipid/lipoprotein abnormalities
- The consequence of increase in total cholesterol
- And It's just not fasting TG
- Mechanisms of hepatic fatty acid to triglyceride
- Fatty acid metabolism
- Regulation of lipolysis
- The antilipolytic effect of insulin
- Response of FFA to insulin/glucose
- Insulin inhibition of FFA turnover
- Insulin action in the liver
- Is there an effect of CHO independent of insulin?
- Effect of SRIF on FFA
- Effect of SRIF on plasma glucose and insulin
- Effect of SRIF on hypertriglyceridemia
- Carbohydrate responsive elements
- SREBP-1c dependent genes in the liver
- ChREBP and SREBP-1c and hepatic lipogenesis
- Plasma VLDL-TG in obese and lean men/women
- VLDL TG kinetics in lean and obese men/women
- Plasma VLDL-TG kinetics in men/women
- VLDL TG clearance in lean and obese men/women
- Adipose tissue LPL responsiveness to insulin
- Patterns of body fat distribution
- Visceral adiposity: the critical adipose depot
- Visceral vs subcutaneous adipose tissue
- Banting lecture 1998
- The clustering of risk factors for ASCVD (2)
- The reduction of HDL cholesterol in obese patients
- Age-adjusted prevalence of HDL cholesterol
- Pathogenesis of decreased HDL cholesterol
- A reason for reductions in HDL cholesterol
- HDL cholesterol and plasma TG
- Plasma levels of HDL particles in the MetS
- Production of HDL particles in the MetS
- Clearance of HDL particles in the MetS
- Pathogenesis of increased atherosclerosis
- The impact of cytokines on obesity and ASCVD
- Inflammatory markers and insulin resistance
- Elevated CRP levels in obese adults
- CRP in overweight and obese men and women
- Inflammation and obesity
- Obesity is associated with cytokine burden
- CRP and body composition
- Visceral adipose tissue/plasma CRP/oxidized LDL
- Changes in metabolic syndrome components
- BMI and CRP in adolescents
- Weight reduction modifies dyslipidemia
- Timing of lipid assessment is important
- Impact of weight reduction on lipids/lipoproteins(1)
- Impact of weight reduction on lipids/lipoproteins(2)
- Impact of weight reduction on plasma triglycerides
- The mechanisms for the reduction in TG in obese
- Molar ratio of VLDL TG/Apo B-100
- Relative change in VLDL Apo B-100 secretion
- VLDL TG secretion: effect of weight reduction
- Impact of weight reduction on HDL cholesterol
- HDL cholesterol,weight reduction and stabilization
- Impact of weight reduction on HDL2 /HDL3
- A diet restricted in total fat lowers HDL and LDL
- Impact of weight reduction on LDL cholesterol
- Change in CRP after 15% weight loss
- Effect of weight reduction on insulin sensitivity
- Effect of weight reduction on plasma hs-CRP
- Effect of weight reduction on interleukin-18 levels
- Gastric bypass and cytokines at 14 Mo
- Summary
Topics Covered
- Normal lipid and lipoprotein physiology
- Abnormalities of lipid and lipoprotein physiology in obesity and related mechanisms
- Inflammation and obesity
- The benefit of weight reduction to favorably modify plasma lipids, lipoproteins and the inflammatory burden in obese patients
Links
Series:
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Therapeutic Areas:
Talk Citation
Eckel, R. (2007, October 1). Obesity, dyslipoproteinemia and inflammation [Video file]. In The Biomedical & Life Sciences Collection, Henry Stewart Talks. Retrieved March 21, 2025, from https://doi.org/10.69645/XMIA3626.Export Citation (RIS)
Publication History
- Published on October 1, 2007
Financial Disclosures
- Prof. Robert Eckel has not informed HSTalks of any commercial/financial relationship that it is appropriate to disclose.