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Printable Handouts
Navigable Slide Index
- Introduction
- Incidence of acute pancreatitis (1)
- Incidence of acute pancreatitis (2)
- Etiology
- Severity of acute pancreatitis
- Pathophysiology of acute pancreatitis (1)
- Pathophysiology of acute pancreatitis (2)
- Pathophysiology of acute pancreatitis (3)
- Pathophysiology of acute pancreatitis (4)
- Diagnosis
- Laboratory analysis
- Radiographs of the abdomen
- Chest X-ray
- Transcutaneous ultrasonography
- Computed Tomography (CT)
- CT Severity Index (CTSI)
- Magnetic Resonance Imaging (MRI)
- MRI (examples)
- EUS of stones & microstones
- Magnetic Retrograde Cholangiopancreatography
- Differential diagnoses of AP
- Criteria for severity prediction
- Ranson criteria
- APACHE II score
- Diagnosis of acute pancreatitis
- 2012: Revision of the Atlanta classification (1)
- 2012: Revision of the Atlanta classification (2)
- 2012: Revision of the Atlanta classification (3)
- 2012: Revision of the Atlanta classification (4)
- 2012: Revision of the Atlanta classification (5)
- The determinant-based classification
- Comparison of classifications (1)
- Comparison of classifications (2)
- Classification of acute pancreatitis
- Outcomes of acute pancreatitis
- Pancreatic exocrine function
- Mortality of acute pancreatitis
- Two peaks of mortality
- World mortality
- Management of acute pancreatitis
- Pharmacological treatment in acute pancreatitis
- Role of ERCP in the management of AP
- ERCP: therapeutic indications
- Early ERCP vs. conservative therapy
- Enteral nutrition in AP
- Is nutrition so important?
- Nutrition in acute pancreatitis (1)
- Nutrition in acute pancreatitis (2)
- Frequency of EN administration
- EN vs. TPN in severe acute pancreatitis (1)
- EN vs. TPN in severe acute pancreatitis (2)
- EN vs. TPN in severe acute pancreatitis (3)
- EN vs. TPN in severe acute pancreatitis (4)
- Comparison of different EN formulations
- Immunonutrition vs. other EN, TPN, or no nutrition
- Probiotic prophylaxis in predicted severe AP
- Probiotics vs. other EN, TPN, or no nutrition
- Nutrition in acute pancreatitis
- Enteral nutrition vs. no nutritional support
- Early vs. on-demand EN administration (1)
- Early vs. on-demand EN administration (2)
- Early vs. on-demand EN administration (3)
- Enteral nutrition in AP
- Antibiotic prophylaxis
- Correlation of pancreatic necrosis and infection
- Prophylactic antibiotics in AP
- Is prophylactic use of antibiotics protective?
- Do we need more meta-analytic studies?
- Infected necrosis in AP
- Infected pancreatic necrosis: early diagnosis
- Minimally invasive techniques
- Percutaneous drainage for pancreatic necrosis
- Endoscopic therapy for pancreatic necrosis
- Laparoscopic technique for pancreatic necrosis
- Retroperitoneal approach for pancreatic necrosis
- Surgical necrosectomy
- Surgery in acute pancreatitis
- The role of early surgery in FAP
- Epidemiology of IAH and ACS in SAP
- Prevention of IAH in patients with SAP
- Nonsurgical treatment of IAH in SAP
- Surgical decompression of IAH in SAP (1)
- Surgical decompression of IAH in SAP (2)
- Pharmacological prevention still impossible
Topics Covered
- Epidemiology and etiology
- Pathophysiology of acute pancreatitis
- Diagnostic methods
- Revision of the Atlanta classification
- Severity prediction
- Outcomes of acute pancreatitis
- Therapeutic options
Links
Series:
Categories:
Therapeutic Areas:
Talk Citation
Štimac, D. (2017, August 31). Acute pancreatitis [Video file]. In The Biomedical & Life Sciences Collection, Henry Stewart Talks. Retrieved March 29, 2024, from https://hstalks.com/bs/1922/.Export Citation (RIS)
Publication History
Financial Disclosures
- Prof. Davor Štimac has not informed HSTalks of any commercial/financial relationship that it is appropriate to disclose.
A selection of talks on Clinical Practice
Transcript
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0:00
The following presentation is a short survey of the basic facts of
Acute Pancreatitis as well as to be introduced to curing Acute Pancreatitis.
0:13
Acute Pancreatitis is a rather common gastroenterological disease with incidence rating
from 5 to 80 cases per 100,000 inhabitants per year.
0:27
As shown in this slide and proven by many national studies,
the incidence of Acute Pancreatitis differs across Europe,
being the highest in the Scandinavian countries namely Finland.
The exact cause is not known but it's probably
related to the differences in the patients genetic traits,
social backgrounds and psychological behavior.
0:55
The etiology of acute pancreatitis is well defined.
Still in around 10 percent of patients,
the cause cannot be determined and these cases are categorized as idiopathic.
In more than 70 percent of the patients,
Acute Pancreatitis is caused either by biliary stones or alcohol consumption.
1:20
In most patients, Acute Pancreatitis is self-limited,
mild to moderate disease.
Complications are rare and death occurs in less than 1% of patients.
Unfortunately, in around 20% of patients,
Acute Pancreatitis follows a more severe course
with the development of Pancreatic Necrosis.
In these patients, infected Necrosis occurs in
30 to 40 percent of cases thus rising the mortality rate up to 30 percent.