Registration for a live webinar on 'Neuroleptic malignant syndrome' is now open.
See webinar detailsProf. Sheila Crowe Division of Gastroenterology UC San Diego, School of Medicine
1 TalkBiography
Dr. Crowe is a Professor in the Division of Gastroenterology in the Department of Medicine at the University of California in San Diego (UCSD). She received her medical degree in 1982 from McMaster University in Ontario, and completed postgraduate training in internal medicine and gastroenterology at McMaster University. After additional... read moreresearch training in the Intestinal Diseases Research Unit at McMaster University and at the University of Toronto, she joined the faculty at McMaster University before moving to the United States in 1992. She spent 9 years at the University of Texas Medical Branch in Galveston where she served as the Gastroenterology Fellowship Training Program Director before moving to the University of Virginia in 2001. There she served as the Director of the UVA Celiac Disease Center, Director of Endoscopy in the UVA Outpatient Surgical Center, Director of the Training in Digestive Diseases T32 Grant, and a member of the Department of Medicine Committee Residency Education. She joined UCSD in July 2011 as the Director of Research in the Division of Gastroenterology and Director of the UCSD Gastroenterology T32 Training Grant. As a clinician-scientist, Dr. Crowe has been active in bench research, clinical care, and teaching in the field of gastroenterology. Dr. Crowe has been named in the Best Doctors in America for her clinical interests in inflammatory bowel disease, gastrointestinal food allergy and celiac disease, and peptic ulcer disease. Her research centers on understanding immune-epithelial interactions involved in inflammatory, infectious, and allergic gastrointestinal diseases including Helicobacter pylori infection. She has received various research grants and career awards and has been funded by National Institute of Health to examine the role of oxidative stress in the gastric mucosa as a mediator of gastric epithelial injury.